Please use this identifier to cite or link to this item: http://gukir.inflibnet.ac.in:8080/jspui/handle/123456789/5547
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dc.contributor.authorRao A.P
dc.contributor.authorPatil S.B.
dc.date.accessioned2020-06-12T15:08:12Z-
dc.date.available2020-06-12T15:08:12Z-
dc.date.issued1993
dc.identifier.citationIndian Journal of Experimental Biology , Vol. 31 , 11 , p. 930 - 931en_US
dc.identifier.urihttp://gukir.inflibnet.ac.in:8080/jspui/handle/123456789/5547-
dc.description.abstractAdult cycling albino rats were hemispayed and administered nicotine for 15 days. FSH/LH or FSH+LH was then administered to these rats. Nicotine inhibited ovarian compensatory hypertrophy significantly and increased cholesterol and lipid levels in the ovary. Administration of FSH alone or in combination with LH restored the ovarian compensatory hypertrophy and decreased the cholesterol and lipid levels significantly, but LH alone was not effective. The results suggest that the inhibition of ovarian growth in nicotine treated rats may be due to lack of availability of pituitary gonadotrophins and these effects can be rectified by the administration of gonadotrophins.en_US
dc.titleEffect of exogenous gonadotrophins on restoration of ovarian activities in nicotine treated unilaterally ovariectomized albino ratsen_US
dc.typeArticle
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